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Fig. 1 | BMC Medical Imaging

Fig. 1

From: Sequential occurrence of eclampsia-associated posterior reversible encephalopathy syndrome and reversible splenial lesion syndrome (a case report): proposal of a novel pathogenesis for reversible splenial lesion syndrome

Fig. 1

Brain magnetic resonance (MR) images for a 23-year-old woman with eclampsia who presented with sudden-onset headache, dizziness, and severe hypertension (blood pressure, 170/110 mmHg). ac. Axial brain MR images obtained at the clinical onset of symptoms show vasogenic cerebral edema in the bilateral parietooccipital lobes, left basal ganglia, and corona radiata. Fluid-attenuated inversion recovery (FLAIR) imaging (a), diffusion-weighted imaging (DWI; b = 1000; b), and apparent diffusion coefficient (ADC) mapping (c) show mild hyperintensity in the lesions (arrows), which is a typical finding in posterior reversible encephalopathy syndrome (PRES). df. Follow-up MR images obtained 10 days after the clinical onset of symptoms (i.e., 1 day after withdrawal of mannitol show that the originally observed hyperintense lesions have disappeared (vasogenic edema has disappeared). FLAIR imaging (d) shows an isolated hyperintense signal in the splenium, whereas DWI (e) and ADC mapping (f) show restricted diffusion in the splenium (arrows), which is a typical finding in reversible splenial lesion syndrome. g–i. Follow-up MR images obtained 29 days after the clinical onset of symptoms FLAIR imaging (g), DWI (h), and ADC mapping (i) show that the lesions in the splenium have disappeared

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